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Compared with diabetic rats, the left ventricular hemodynamic parameters and myocardial ultrastructure changes (myocardial hypertrophy and fibrosis) are obviously normalized in diabetic animals treated with NaHS, which may be associated with upregulation of cardiac myosin light chain kinase (MLCK) and downregulation of serum creatine kinase MB isozyme (CK-MB) (Yang et al., 2016a)

To further confirm the findings, pseudotime analysis revealed that clusters expressing high monocyte markers served as root cells, and macrophages differentiated through the intermediate cluster 3 to produce the activated mature innate immunologic cells or the mature alveolar macrophages (Fig

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doi: 10.1038/nrc3495 90 SongJLiuTYinYZhaoWLinZYinYet al